Additionally, ninjurin2 shares 55% of its homology with another component of the same family, ninjurin1 (NINJ1), which, besides playing a role in promoting axonal outgrowth in the nervous system after nerve damage [9], has also been implicated in the adhesion and migration of myeloid cells through the BBB in a murine model of MS [28,29]. This evidence concerns the gene NINJ1 and myeloid sarcoma.