In line with our previous results showing that Glutor treatment can induce an upregulation of GLUT-3 in tumor cells [12], our transcriptional analysis demonstrates that NK cells try to counteract the inhibited uptake of glucose by increasing the expression of GLUT-3 (SLC2A3), GLUT-13 (SLC2A13) and GLUT-14 (SLC2A14), as well as other genes related to glycolysis. Here, SLC2A14 is linked to neoplasm.