RBFOX1 and early-onset autosomal dominant Alzheimer disease: The second assumption is based on several studies implicating Rbfox1 in the stress-induced regulation of several mechanisms that stimulate cell survival, including miR-132/Rbfox1-mediated mechanisms that promote neuronal survival against amyloid β-peptide (Aβ) and glutamate excitotoxicity in the brain of the mouse model for Alzheimer’s disease [26,39]