NSUN2 and Alzheimer disease: We did not observe any decrease in the levels of NSun2 in the tissues from the prefrontal cortex, hippocampal formation and cerebellum (PART versus controls; PSP versus controls) (Fig. 2a, b, d, e, c, g) and from the globus pallidus (PSP versus controls) (Fig. 2f), suggesting that the reduction of NSun2 is a selective feature of AD brains when compared to other tauopathies.