In the non-insulin-resistance model, after adding drugs to incubate L6 cells, the effect of 60 μg/mL AP-EtOAc on the uptake of glucose was significantly higher than that in the control group, and the uptake of glucose effect of 90 μg/mL AP-EtOAc was comparable to that of insulin. The gene discussed is INS; the disease is Insulin resistance.