LPA and familial hyperaldosteronism: 74 patients with FH (8.2%) were reclassified to unlike FH when LDL-C was corrected for Lp(a)-cholesterol.There were no significant differences detected in the proportion of pathogenic FH mutations (27.9% vs. 33.1%) between patients with increased and normal Lp(a) concentrations at a cutoff of 50 mg/dL (p = 0.05).