Of note, CD84, CD148, CD197, and CD261, as well as all other genes that significantly discriminated between two AML subgroups only in one dichotomy (APL vs. AML1, APL vs. AML2, AML1 vs. AML2), were only found in one dataset and showed a Cohen’s D-value of 0 in the other and CD91 was not expressed in the microarray dataset (Table S1). Here, LRP1 is linked to acute promyelocytic leukemia.