Epstein–Barr virus-associated gastric cancer (EBVaGC) was one of four classifications of GC, with frequent mutations of PIK3CA, ARID1A, and BCOR, and PD-L1 and PD-L2 amplification (4); it accounts for up to 10% of its molecular subtypes (5). This evidence concerns the gene PDCD1LG2 and gastric cancer.