There was no significant difference in the proportion of hypertension history between patients with SAH and patients with SAH + CVS (all p > .05); the blood glucose, blood sodium fluctuation, serum lactic acid, IL‐6, ET‐1, and PDGF were raised in patients with SAH and patients with SAH + CVS compared with healthy subjects, and upregulated in patients with SAH + CVS compared with patients with SAH (all p < .05, Table 2). The gene discussed is EDN1; the disease is hypertensive disorder.