An increase was also detected for proteins involved in coagulation (F5, F9, F10, F11, PROS1, SERPIN C1, SERPIN A10, SERPIN F2, HABP2) inflammation-induced proteins involved in different types of amyloid diseases (SAA1, SAA2-SAA4, APCS, APMAP, B2M, CST3) and extracellular matrix organization (FN1, VTN, ITH3, ITH4). This evidence concerns the gene B2M and amyloidosis.