In rescue experiments, the upregulation of CXCL3 bolstered the malignant phenotype of cervical cancer cells in proliferation (Figure 3(e), p < 0.001), migration (Figure 4(a), p < 0.001), and invasion (Figure 4(b), p < 0.001) compared to its control group, which also starkly reversed the antitumor effect of solamargine on cell proliferation (Figure 3(e), p < 0.001), migration (Figure 4(a), p < 0.01), and invasion (Figure 4(b), p < 0.01). The gene discussed is CXCL3; the disease is cervical carcinoma.