Wang et al. reported that the long noncoding RNA RNM3OS inhibited miR-361 and miR-29a/29b, which suppressed the expression of COL3A1 and TGF-β1, leading to BPH development by promoting TGFβ1-induced transformation of prostate stromal cells into myofibroblasts 30. The gene discussed is TGFB1; the disease is benign prostatic hyperplasia.