Single‐nucleotide polymorphisms purported to be associated with PD include ones in the clock genes CLOCK; BMAL‐1; PER1,2; and CRY1,2, and it has been proposed that alterations in clock genes might contribute to PD pathogenesis through alteration of circadian modulation of process such as mitochondrial bioenergetics, autophagy and alteration in neuroendocrine function (Shkodina et al., 2022). This evidence concerns the gene CRY1 and Parkinson disease.