Because Tip60 is pro-apoptotic (Tang et al., 2006; Sykes et al., 2006), it was of interest to assess whether treatment with TH1834 on days 3-16 post-MI reduced apoptosis, as well as myocardial scarring due to infarction, in hearts removed for histological inactivation at days 10 and 28 post-MI. The gene discussed is KAT5; the disease is myocardial infarction.