Comprehensive analysis of the GO and KEGG enrichment results (Figures 6(c) and 6(d)) revealed that key target genes have cytokine, chemokine, and signal receptor activator activities, activate cytokines and chemokines through NF-κB, TNF, and IL-17 signaling pathways, promote CXCR chemokine or cytokine binding to receptors, and participate in a variety of inflammatory or immune responses, including neutrophil chemotaxis and migration, rheumatoid arthritis or parasitic infection, and alcoholic liver disease. This evidence concerns the gene NFKB1 and parasitic infectious disease.