However, all data indicated that there was no binding site in the promoter regions of both Cyclin D1 and c-Myc for KLF13 (data were not shown), which demonstrated that these two genes were not direct transcriptional targets of KLF13.These data revealed that KLF13 suppressed cell proliferation through inhibiting β-catenin expression and its downstream genes, CCND1 and MYC, in gastric cancer. The gene discussed is KLF13; the disease is gastric cancer.