Activity of another transcription factor - AhR, was also found to be suppressed upon MEN1 silencing, whose role in controlling proliferation, migration, and tumour cell invasion has not been extensively determined yet, however there are some indications for its tumour-promoting role in OC since AhR nuclear localization has been associated with worth outcome for OC patients [48] and evidences of AhR receptor role in promotion of cell growth, stemness and metastatic potential of OC [49]. This evidence concerns the gene MEN1 and neoplasm.