In addition, the genotypes of thalassemia disease were also observed, including one case each of the EABart’s disease or Hb H disease with Hb E heterozygote (–SEA/− α3.7, βΑ/βΕ), Hb E-β+-thalassemia disease (αα/αα, β−28/βΕ), and Hb E-β+-thalassemia disease with heterozygous α+-thalassemia (αα/-α3.7, β−28/βΕ). This evidence concerns the gene GSTM1 and glycogen storage disease VI.