Western blots with anti-pY701STAT1 antibodies and Phospho-STAT1 flow cytometry show that suppression of TC-PTP activity with compound #182 (19) after IFN-γ stimulation in NCI-H358 lung cancer cells led to increased STAT1 Y701 phosphorylation (Fig. S1). The gene discussed is IFNG; the disease is lung carcinoma.