Since in our dual-infection scheme, we wanted to apply the second virus to a cell population that consists of the subpopulation of the preinfected and IFN-producing cells and the subpopulation of not (yet)-infected but IFN-stimulated cells, we primed cells with the first virus, RSV, at an MOI of 0.1, and after 30 h, we applied the second virus, IAV, at an MOI of 1. This evidence concerns the gene IFNA1 and infection.