Early studies showed that activated neutrophils from cancer patients suppress T‐cell function through release of H2O2.98 This suppressive capacity of neutrophils on T‐cells occurs in humans during LPS‐induced acute inflammation, dependent on bacterial opsonization through neutrophil Mac‐1 (integrin αMβ2; CD11b/CD18), and subsequent H2O2 release leading to suppression of T‐cell proliferation.4 The gene discussed is ITGAM; the disease is cancer.