IFNB1 and neoplasm: The induction of TAN pro‐inflammatory N1 and anti‐inflammatory N2 subsets is dependent on the tumor microenvironment, and specifically, it is linked to TGF‐β promoting the cell differentiation toward ab anti‐tumor N1 neutrophil phenotype,42 while IFN‐β favors a pro‐tumor N2 subset43 (reviewed in 44).