In our rat models we could demonstrate that the T cell populations within the eyes during uveitis are highly dynamic, they change their phenotype by co-expressing IFN-γ and IL-17, even together with IL10, indicating that they might convert to Tregs, since the IFN-γ/IL-17/IL-10 co-expressing cells increase at the resolution of the monophasic disease and decrease during the course of the relapsing uveitis. The gene discussed is IL17A; the disease is uveitis.