Hb levels in the CDDP-administered anemic mice (120 g/L) were up-regulated by TBN to close to the normal levels (160 g/L), performing on par with the positive drug Roxadustat, and treatment with TBN stopped the development of CDDP-induced anemia (Figures 2C–E). This evidence concerns the gene GSTM1 and anemia (phenotype).