NFKB1 and experimental autoimmune encephalomyelitis: In experimental autoimmune encephalomyelitis (EAE) mice and lipopolysaccharide-induced BV2 cells, Yue and colleagues discovered that down-regulation of TUG1 attenuates MS through the inhibition of inflammation by sponging miR-9-5p via targeting NF-κB1/p50 (Yue et al., 2019).