PSEN1 and sleep apnea syndrome: However, the number of cBF neurons in APP/PS1 (Fig. 9B; and wildtype Supplementary Fig. 3E) high oxygen-treated cMPT-lesioned animals was significantly higher than that of untreated lesioned mice, being similar to that of sham-lesioned animals in standard housing (Fig. 9B), thereby confirming that the cBF degeneration was a result of the SDB phenotype.