Myeloid cells (CD14, CD16, and CD86 nobocytes) were not clustered or differentially regulated, except for a reduction of CD57+ CD86 monocytes in patients with ALS compared with that in HCs in manual gating only (p < 0.05; eFigure 4, links.lww.com/NXI/A750). This evidence concerns the gene CD14 and amyotrophic lateral sclerosis.