FOXM1 and meningioma: The remaining meningiomas were distinguished by enrichment of pathways associated with lipid or nucleotide metabolism, termed “Hypermetabolic”, or “Proliferative” and “Hypermitotic” meningiomas that were enriched in cell cycle and proliferative gene programs such as the FOXM1 program, and associated with near uniform loss/inactivation of NF2, genomic instability with accumulation of high risk CNVs such as 1p loss.