Interestingly, several intermediates including R5P, Xu5P, G3P, glucose, and α‐ketoglutarate were markedly accumulated in oroxylin A‐treated cells and tumours (Figure 3B–E), suggesting activity inhibition of TKT by oroxylin A. Therefore, activity suppression of TKT by oroxylin A led to accumulation of glycolytic intermediates in the non‐oxidative PPP (Figure 3F). Here, TKT is linked to neoplasm.