Propofol inhibited the expression of STAT3 by upregulating miR-125b-5p in gastric cancer cells, and STAT3 overexpression reversed the accumulation of Fe2+, ROS, MDA levels in erastin-treated SGC7901 and BGC823 cells by propofol, thereby demonstrating that propofol can enhance the ferroptosis of gastric cancer cells (Liu et al., 2021a). Here, STAT3 is linked to gastric cancer.