Although it is unknown whether AUR inhibits MsF1, this difference in composition could be useful for the development of species-specific antimicrobial drugs to treat tuberculosis and other communicable diseases such as brucellosis (Brucella sp), botulism (Clostridium botulinum), cholera (Vibrio cholera), and diphtheria (Corynebacterium diphtheriae). This evidence concerns the gene SEPTIN9 and botulism.