TSLP and its isoforms may contribute to the Kawasaki Syndrome seen in SARS-CoV-2 infected children, or to increased thrombosis in COVID-19 severe cases, through activation of vascular endothelial cells and/or platelet activation via TSLP-dependent PI3K/Akt signaling 57-59. This evidence concerns the gene TSLP and Kawasaki disease.