More recently,SARS-CoV-2 has also been shown to bind to LSECtin,an interaction proposed to be on cargo of a specific GlcNAc terminatingN-glycosylation site and interfering with the ACE2/spike interaction.18 Moreover, the presence of LSECtin enhances thebinding of Lassa virus to host cells and enhances the infection oflymphocytic choriomeningitis virus (LCMV).19,20. The gene discussed is CLEC4G; the disease is infection.