NFKB1 and tuberculosis: miR-223 is abundantly expressed in monocytes and monocyte-derived macrophages (MDMs) from TB patients and attenuates nuclear translocation and p65 (a subunit of NF-κB) (66) phosphorylation, resulting in suppression of NF-κB activation and inhibition of cytokine secretion, hindering M. tuberculosis eradication (57).