Meanwhile, another study pointed out a marked increase in hsa-miR-337-3p and hsa-miR-125b-5p in patients with TB is causative of impaired STAT3 function, which results in the abrogation of IL-23-mediated expansion of Vγ2Vδ2 T cells and weakens the ability of these cells to produce anti-TB factors (52). The gene discussed is STAT3; the disease is tuberculosis.