This protein may also serve as an ebolavirus attachment factor as EBOV GP binding to dendritic cells is partially abrogated by anti-LSECtin polyclonal antiserum and because transfection-induced LSECtin expression in a chronic myelogenous leukemia cell line enhanced EBOV GP-pseudotyped lentivirus entry (Dominguez-Soto et al., 2007). Here, CLEC4G is linked to chronic myelogenous leukemia, BCR-ABL1 positive.