Interestingly, the role of CD36 in the resolution of neuroinflammation in different noninfectious models has been previously reported, such as in Alzheimer’s disease, stroke, ischemic brain due to middle cerebral artery occlusion, brain hematoma, or germinal matrix hemorrhage (Ballesteros et al., 2014; Woo et al., 2016; Zhang et al., 2018; Zhao et al., 2020; Davaanyam et al., 2021; Dobri et al., 2021). The gene discussed is CD36; the disease is stroke disorder.