Considering the high expression of CaMKK2 in TAMs (Fig. 1b and Supplementary Fig. 2d), TAM abundance in the TME (Fig. 2d and Supplementary Fig. 2b), and the immunostimulatory impact of CaMKK2 deficiency on TAMs, we conditionally deleted CaMKK2 in MNPs (LysMcre x CaMKK2fl/fl) to determine whether this would be sufficient to produce a survival benefit in IC tumor-bearing mice. Here, CAMKK2 is linked to neoplasm.