We specifically evaluated the role of AhR in controlling cell migration by performing wound‐healing assays of BRAFi‐sensitive or resistant melanoma cells (called SK28S and SK28R, respectively) after genetic depletion of AhR (SK28S/R KO; Fig 1B) or chemical inhibition using its specific antagonist (CH‐223191, 10 μM; Fig 1C). The gene discussed is AHR; the disease is melanoma.