It was demonstrated that elevated Lp(a) concentrations were independently associated with an increased risk of abdominal aortic aneurysms (AAA) in a population-based cohort study [36], and a meta-analysis demonstrated that high levels of Lp(a) may be linked to the presence of AAA and that Lp(a) may be a marker to screen for AAA [37]. This evidence concerns the gene LPA and abdominal aortic aneurysm.