The role of Lp(a) in venous embolic events such as pulmonary embolism(PE) is controversial, potential pathogenic mechanisms of Lp(a) include its similarity to fibrinogen, leading to reduced fibrin synthesis and fibrinolysis inhibition, the tendency of Lp(a) to oxidize upon entry into the vessel wall, and the production of highly immunogenic and pro-inflammatory phospholipids [16, 17], and whether these effects play an important role in PE is unclear [18]. Here, LPA is linked to pulmonary embolism.