The finding was also supported by Wei et al who have demonstrated that KLF5 deacetylated by HDAC4 potentiates the airway inflammation and remodeling in asthma through enhancing Slug and CXCL12 expression20 and Lu et al. who have explicated that HDAC4 targeted by miR‐20a‐5p contributes to allergic inflammation in human mast cells.21 This evidence concerns the gene SNAI2 and asthma.