C/EBPβ expression is inhibited in diabetic cardiomyopathy- (DCM-) induced fibrotic heart tissue, and overexpression of C/EBPβ inhibits this cardiac fibrosis, while C/EBPβ knockdown attenuates heart fibrotic pathology in rat models of experimental autoimmune myocarditis (EAM) [34, 38]. Here, CEBPB is linked to diabetic cardiomyopathy.