IFNG and infection: Indeed, when we removed the sample-specific variability by calculating the IFN-γ response as a percent of untreated control infection for each sample, we observed statistically significant correlations between the inhibitory effect of IFN-γ and the total GFP positive B cells in the untreated conditions with a monotonic relationship between the variables, thus statistically validating the observation that susceptibility to KSHV infection is related to responsiveness to IFN-γ treatment (Figure 1c).