Such is the case of community-acquired strains 139 (respiratory infection) and 140 (UTI), which presented the same genotype (fimH/kpn/mrkD/ycfM/entB/irp1/irp2/ybtS) and phenotype (HM-positive, moderate biofilm formation, and multidrug resistance), and were both non-K1/K2. Here, ACO1 is linked to respiratory tract infectious disorder.