These clinical symptoms require accompaniment by laboratory findings of inflammation such as erythrocyte sedimentation rate (ESR), C-reactive protein (CRP) or procalcitonin (PCT) levels [19], and evidence of SARS-CoV-2 infection or contact with COVID-19 patients ruling out bacterial sepsis, staphylococcal or streptococcal shock syndromes caused by infection with other pathogens [3]. This evidence concerns the gene CRP and infection.