LPA and coronary artery disorder: Lp(a) is associated with risk for CAD through multiple mechanisms (Figure 1) including atherogenesis mediated by apoB [12], vascular inflammation mediated by its carriage of oxidized phospholipids (OxPL) [13,14,15,16], and anti-fibrinolytic effects that may be related to the homology of apo(a) with plasminogen [17].