This ultimately leads to hyperglycemia (glucose not being sequestered at the cellular level, and rather remaining in circulation) and hyperinsulinemia (the pancreas, feeling that hyperglycemia is present, is stimulated to produce new insulin), and these two situations are potentially one oncological bomb, being capable of intervening both directly and/or indirectly in the transformation and progression of a breast cell in oncology [9]. Here, INS is linked to Hyperinsulinemia.