While there are contradictory studies on the function of NEAT1 in CML, no studies have yet explored the role of CCAT2 in CML, although one has shown lncRNA CCAT2 is dysregulated in CML, with unusually high expression in the CP of CML compared to healthy controls. Here, CCAT2 is linked to chronic myelogenous leukemia, BCR-ABL1 positive.