In total, our results indicate that between episodes of rhabdomyolysis, there is ongoing molecular evidence of an oxidative stress response exemplified by the differential expression of genes and proteins involved in the production and maintenance of antioxidants with notable alterations in protein and gene expression for thiol-based peroxiredoxins, thioredoxins and precursors for glutathione synthesis. This evidence concerns the gene TXN and rhabdomyolysis.