Additional morphofunctional abnormalities were recently reported: for instance, a direct correlation was shown between the LV/RV dilation and SCN5A mutation, with wider involvement of the RV than the LV [54] as observed in the classic arrhythmogenic right ventricular cardiomyopathy (ARVC). This evidence concerns the gene SCN5A and Arrhythmogenic right ventricular dysplasia.