Additional morphofunctional abnormalities were recently reported: for instance, a direct correlation was shown between the LV/RV dilation and SCN5A mutation, with wider involvement of the RV than the LV [54] as observed in the classic arrhythmogenic right ventricular cardiomyopathy (ARVC). Here, SCN5A is linked to arrhythmogenic right ventricular cardiomyopathy.