Saleh et al. reported that a relatively high percentage of patients with COVID-19 with no known history of CVD had elevated cardiac damage and inflammatory biomarkers such as cardiac troponin, N-terminal pro-brain natriuretic peptide (NT-proBNP), and lambda immunoglobulin free light chains (Ig FLC) suggestive of myocarditis [13,14,15]. The gene discussed is NPPB; the disease is COVID-19.