This is not only simply because oxidative stress generated by dysregulated copper may induce cell transformation and uncontrolled proliferation (as the increased level of copper found in tumors of different origin or in the serum of cancer patients may suggest) but, more intriguingly, because this metal was found to bind and regulate the function of protein kinases, acting as positive or negative allosteric regulators. This evidence concerns the gene WEE1 and cancer.